BOMBSHELL Study: Covid ‘Vaccines’ Alter Human Behavior
Proof that the covid-19 vaccine causes mental illness
Leading Cardiologist: 100 Million Vaccinated Americans May Have IRREVERSIBLE Heart Damage
Bombshell Study Exposes Toxic Effects of Covid mRNA ‘Vaccines’
FDA ‘s Process of Drug and Vaccine Evaluation, Part I
HORRIFIC: Heart Attacks in 2-Year-Olds…Courtesy of the Clot Shots
HOW DID THEY SAY COVID VACCINES WERE “SAFE”?
Another trick you need to know about
Nothing to see here …
From Mark Crispin Miller @ Substack
Sheffield Eagles full-back Quentin Laulu-Togaga’e “recovering from heart attack”; footballer Tom Holmes “diagnosed with rare form of cancer”; ITV soap “Emmerdale” adds cancer storyline
READ MORE ….
More celebrities sidelined dramatically in just the last few days—and a sampling of the “rare”conditions now afflicting countless tots and babies (none famous, or related to celebrities)
Indianapolis Colts owner Jim Irsay undergoing treatment for “severe respiratory illness”
These first two items reconfirm what we (who pay attention) have unhappily observed since early 2020—that cultural rebelliousness (Stern’s raunchy humor, RATM’s loud dissidence) does not make you anti-authoritarian (if you’ve been stupefied with fear).
Though he’s never felt sicker, Stern credits the “vaccine,” without which he’d feel even worse, he thinks (if one can use that verb with so feral a Covidian).
READ MORE ….
An absolute must hear. Dr Miller gives an overview of the democide as he calls it, to date. His life he says is now devoted to awakening people to what is really happening globally. It is very evil…. TWNZ
From Dr. Mark Crispin Miller @ Substack
Join Greg Hunter as he goes One-on-One with NYU Media Studies Professor and propaganda expert Dr. Mark Crispin Miller for 12.16.23.
To Donate to USAWatchdog.com Click Here: https://usawatchdog.com/donations/
https://substack.com/@markcrispinmiller1
LISTEN AT THE LINK
https://rumble.com/v41jmgt-cv19-vax-was-rolling-thunder-global-propaganda-mark-crispin-miller.html
From hatchardreport.com
The Dentist:
My dentist friend received mRNA COVID-19 vaccines in 2021. He and his partner developed cardiac issues, both have since struggled to recover their full health. They have been alert and informed enough to examine published evidence and concluded the vaccine was likely at fault.
This article is also available as a PDF document to print/download and share.
His subsequent experience at his dental practice paints a picture of vaccine harm that is sobering and truly frightening. He has 2200 patients; during the last two years, a great number of them have reported new serious health conditions as follows. These figures only include accounts that his patients raised with him in order to seek specific advice or help. It doesn’t include the many who suffered in silence.
Clinically recognised cardiac issues – 69 (not counting the significant numbers who have told him they are struggling but not having sought help).
Stroke – 5.
Cancer – 20. (Pancreatic cancer stands out in incidence).
Blood clots – 3.
Sudden death – 11.
READ AT THE LINK
https://hatchardreport.com/what-the-dentist-saw-and-what-the-judge-thought/
photo: hatchardreport.com
From mercola.com
Can you be fat and still be fit? An estimated 15% to 20% of obese individuals have none of the metabolic derangements associated with obesity, such as high blood sugar, high blood fats, high blood pressure, Type 2 diabetes and other risk factors for cardiovascular disease.1
This phenomenon has led to the idea that you can be obese yet in good health. But according to recent research,2,3,4 this popular belief — known as “metabolically healthy obesity” (MHO) — is a fallacy. As reported by studyfinds.org:5
“The myth of being ‘fat but fit’ is, again, being debunked by a new study. Researchers found that obese individuals, even if they appear healthy, have an increased risk of diabetes and … are still 50% more susceptible to coronary heart disease.”
No Such Thing as ‘Fat and Fit’
According to professor Matthias Blüher, who presented the findings at the 2023 Meeting of the European Association for the Study of Diabetes,6 high body mass index (BMI) in combination with normal glucose and lipid levels, and the absence of high blood pressure, is not sufficient for MHO classification. Rather, it’s the behavior of the adipose cells that matters.
People who have normally sized adipocytes (cells that store fat), have far fewer obesity complications compared to those who have enlarged and inflamed adipocytes.
Dysfunctional adipocytes can result in fibrosis and the release of harmful molecules that contribute to organ damage. Fat-secreted hormones, known as adipokines, can also directly affect cells in your vascular system, leading to atherosclerosis.
Where you store most of your fat may also be key. Obese individuals with large visceral fat stores, meaning the fat is primarily stored around their internal organs, are far more likely to develop Type 2 diabetes than those whose fat stores are distributed more evenly around their body, the study found. Other studies have also confirmed this.7
What this means in practical terms is that obese patients still need to focus on losing weight and may need other preventive treatments as well. In an October 3, 2023, press release, Blüher explained:8
“So there is still a residual increased risk for those people living with obesity, even with what we would call metabolically healthy obesity.
Even in the absence of other cardiometabolic risk factors, increased fat mass and adipose tissue dysfunction contribute to a higher risk of Type 2 diabetes and cardiovascular disease. Therefore, weight management and recommendations for weight loss are still important for people living with metabolically healthy obesity.”
Other Studies Concur
Other recent research has come to the same conclusion. A prospective cohort study of 381,363 British participants, published in 2021, found that:9
“Compared with people who were not obese at baseline, those with MHO had higher incident HF [heart failure] (HR [hazard ratio] 1.60 …) and respiratory disease (HR 1.20 …). The associations of MHO were generally weaker for fatal outcomes and only significant for all-cause (HR 1.12 …) and HF mortality rates (HR 1.44 …).
However, when compared with people who were metabolically healthy without obesity, participants with MHO had higher rates of incident diabetes (HR 4.32 …), ASCVD [atherosclerotic cardiovascular disease] (HR 1.18 …), HF (HR 1.76 …), respiratory diseases (HR 1.28 …) and all-cause mortality (HR 1.22 …) …
Conclusions/interpretation: Weight management should be recommended to all people with obesity, irrespective of their metabolic status, to lower risk of diabetes, ASCVD, HF and respiratory diseases. The term ‘MHO’ should be avoided as it is misleading and different strategies for risk stratification should be explored.”
In the Energy Balance Podcast above, independent health researchers Jay Feldman and Mike Fave explain why eating too much and exercising too little is not the reason for weight gain. Jay is one of the best teachers of the Bioenergetic view of health. I would strongly encourage you to listen to his YouTube channel, but watch the oldest ones first so you can get grounded in the basics.
Obesity is a state of energy DEFICIENCY due to inhibited mitochondrial respiration, which causes calories to be stored as fat instead of being burned for fuel. The solution, therefore, is not to eat less and exercise more. Rather, it’s to optimize your mitochondrial function and raise your metabolic rate.
This inefficient burning of fuel (metabolizing of food) is why people who are obese typically also struggle with other health issues, such as low energy, fatigue, an inability to maintain focus, digestive problems and poor immune function.
As explained by Feldman, “energy,” in terms of the human body, primarily refers to your ATP level. So, ATP equals energy. The conventional view is that calories equal energy, but that’s a misconception, because calories come from three primary substrates (fat, glucose and protein), and they do not produce the same amount of ATP calorie for calorie.
Another major misconception is that when you’re gaining weight, you’re converting all your fuel (from food) into ATP and simply don’t need any more (typically because you’re not active enough), and so the excess energy is stored as body fat instead of being burned.
What actually happens is that you have a problem converting the fuel you consume into energy, so the fuel gets stored as body fat instead. In other words, you’re not producing enough energy and are in an energy-deficient state. So, it’s not excess ATP/energy that gets stored. It’s the fuel that is stored, and it’s stored because your body cannot efficiently metabolize it.
The end result is excess body fat AND insufficient energy, which in turn forces your body to downregulate other systems, such as your thyroid activity and reproductive hormones — anything that is not essential for survival.
You’re also cursed with perpetual hunger because the hunger signal is predominantly regulated by energy availability. This in turn leads to overeating, resulting in a vicious cycle of low energy and weight gain.
A study10 was recently published in the International Journal of Molecular Sciences, attributing obesity to broken mitochondria. As noted by the authors:
“An important component of the pathogenesis of the metabolic syndrome is mitochondrial dysfunction, which is associated with tissue hypoxia, disruption of mitochondrial integrity, increased production of reactive oxygen species, and a decrease in ATP, leading to a chronic inflammatory state that affects tissues and organ systems.”
In the podcast, Feldman cites an earlier study, published in 2003 in the Journal of Theoretical Biology, titled “Decreased Energy Levels Can Cause and Sustain Obesity.” An excerpt from the abstract reads:11
“Current theories consider obesity a result of overeating and sedentary life style and most efforts to treat or prevent weight gain concentrate on exercise and food intake. This approach does not improve the situation as may be seen from the steep increase in the prevalence of obesity.
This encouraged us to reanalyze existing information and look for biochemical basis of obesity … We began with the contradictory information: in obesity, more calories are consumed than used up, suggesting that obese people should have excess energy.
On the other side, obese people experience fatigue and decreased physical endurance that indicates diminished energy supply in the body. The result of our work is a chain of metabolic events leading to obesity.
The crucial event is the inhibition of the TCA [tricarboxylic acid] cycle at the step of aconitase. It disturbs energy metabolism and results in ATP deficiency with simultaneous fat accumulation.
Further steps in obesity development are the consequences of diminished energy supply: inhibition of beta-oxidation, leptin resistance, increase in appetite and food intake and a decrease in physical activity.
Thus, our theory shows that obesity does not have to be caused by overeating and sedentary life-style but may be the result of the ‘obese’ change in metabolism which is forcing people to overeat and save energy to sustain metabolic functions of cells. This ‘obese’ change is caused by environmental factors that activate chronic low-grade inflammatory process in the body …”
The screenshot below can be helpful. On the left is what normal metabolism looks like. The food you eat is turned into fuel, some of which is burned for energy and some of which is stored as body fat. When needed, body fat is also burned as fuel and converted into energy.
On the right is the “obese” metabolism. The primary differences here are the dotted arrows. The food consumed is turned into fuel, but very little of that fuel is converted into energy. Most of it is instead shuttled into fat storage, and because the metabolism is so inefficient, the stored fat is not released and burned for energy.
As explained by Feldman, not only do obese individuals have this deranged metabolism, but many normal weight people do too. People who exercise a lot, or fast, or eat a low-carb diet may appear to have healthy metabolism because they’re not overweight, but the real reason they’re not overweight is because they’re forcing the conversion of fuel to energy through STRESS.
So, while they may not accumulate body fat, they still struggle with an energy deficiency, which can present as fatigue, brain fog, low mood, hormonal dysfunction and more.
As noted by Feldman, the bioenergetic solution to these problems is to improve the conversion of fuel to energy, “which means you have the energy available to properly function in terms of all your organ systems — your digestion, your reproductive systems, cognitive function, all of that.”
One key strategy to optimize your mitochondrial energy production is to remove blocks in the electron transport chain so that electrons can move smoothly forward, without accumulating and backing up.
Endotoxin (lipopolysaccharide or LPS) and other bacterial toxins are among the biggest culprits when it comes to things that hinder mitochondrial energy production. These toxins can directly impair electron transport through the complexes of the electron transport chain. They can also impair certain enzymes in the Krebs cycle.
The primary solution for obesity and most other conditions is to raise your metabolic rate, and a key strategy for that is to remove blocks in the electron transport chain. Endotoxin and polyunsaturated fats (PUFAs) are two key culprits that need to be eliminated.
Poor digestion is frequently associated with negative gram bacteria in your gut that produce endotoxin, and this will inhibit your ability to convert food to energy, resulting in increased body fat. So, it’s important to reduce your endotoxin load. Aside from poor digestion, excess endotoxin is also a common culprit in degenerative conditions, metabolic syndrome, diabetes and fatty liver. To reduce your endotoxin load:
Once your microbiome is balanced and symptoms of poor gut function have resolved, you can slowly reintroduce starches like white rice and, if tolerated, progress to very well-cooked vegetables. Always cut back if you have signs of indigestion like belching, bloating or gas as this suggests you are not digesting those carbs yet.
Another effective blocker of mitochondrial energy production is polyunsaturated fat (PUFA). These are your processed seed oils, canola oil being among the worst of the worst. Seed oils are loaded with linoleic acid, an omega-6 PUFA, which appears to be one of the primary drivers of chronic diseases, in part due to its detrimental impact on your mitochondrial function and energy production.
So, you’ll want to severely limit your consumption of PUFAs. In addition to not cooking with them, this also means abstaining from processed foods, most of which are loaded with these harmful fats.
While there are no magic pills to fix slow metabolism or low energy production, there are some that can help. I’ve previously written about the usefulness of niacinamide, for example. Another helpful one is methylene blue, which can both accept and donate electrons.
So, if you have blocks in the electron transport chain where most of the ATP is produced, say endotoxin or PUFAs, methylene blue can bypass those blocks, allowing your mitochondrial to produce energy anyway.
According to the rate-of-living theory, the higher your metabolic rate — which means the quicker the electrons move from food toward oxygen, which is the final acceptor of electrons — the faster you’ll age because there’ll be higher oxidative stress.
However, deeper analysis reveals the exact opposite. The truth is, the higher your metabolic rate, the slower you age, because a high metabolism creates fewer reactive oxygen species (ROS) that can damage your tissues.
Your metabolism is high when electrons move rapidly and easily through the mitochondrial electron transport chain, which results in optimal energy production. When electrons are impeded from moving forward, they can back up, leak through the mitochondrial electron transport chain and start moving backward, where they combine with oxygen to create excessive ROS.
So, for optimal health, you want high energy production and that means a high metabolic rate. You can gauge your metabolic rate using your pulse, body temperature and calorie counting. These are not exact sciences, but can give you a general idea of where you’re at. Signs of low metabolism include:
Sources and References
Saturday, July 7th 2018 at 10:45 am
Originally published on www.vaccineimpact.com
Two leading pediatricians in India have published a critique of the World Health Organization’s (WHO) newly revised manual on the classification of Adverse Events Following Immunization
The doctors claim that the new guidelines are too lax, resulting in more children dying from adverse reactions to vaccines, with no accountability for the vaccine manufacturers to produce safer vaccines.
In the U.S., pharmaceutical companies manufacturing vaccines cannot be sued due to faulty vaccines, which allows them to put as many vaccines into the market as they want, with no repercussions if the vaccine proves to be faulty or have a high injury and death rate. Victims must sue the U.S. government and top DOJ attorneys who protect the drug companies. (See quarterly reports from the DOJ on vaccine injuries and deaths here.)
This move by the WHO may very well be an attempt to protect drug companies manufacturing vaccines by preventing lawsuits in the rest of the world for faulty vaccines.
New Guidelines from WHO Put Children’s Life At Risk: Paediatricians
Two leading paediatricians in India have urged the World Health Organization (WHO) to urgently revise its manual on classification of Adverse Events Following Immunization (AEFI), warning that the new guidelines put children’s life at risk.
This needs to be done “urgently in the interest of child safety,” Doctors Jacob Puliyel at St Stephen’s Hospital in Delhi, and Pathik Naik of Children Hospital in Surat, say in a report published in the prestigious journal ‘F1000Research‘
Under WHO’s revised manual on AEFI, only those adverse reactions observed during clinical trials of a vaccine, should be classified as vaccine related. All new serious adverse reactions, including deaths seen during post-marketing of the vaccine should be considered as ‘coincidental’ or ‘unclassifiable’, and the vaccine should not be blamed.
The WHO has also changed the definition of “causal association,” the doctors say. Under the revised guidelines, if there is an alternate explanation for the adverse event, or another factor is involved, causative association with vaccine should not be made.
“In other words, if after vaccination, a child with an underlying congenital heart disease develops cardiac failure, it would not be considered causally related to the vaccine.”
The revised classification by WHO “is a major step backward for patient safety,” the authors say, adding, “This could embolden vaccine manufacturers to be more reckless with regard to adverse reactions.”
Puliyel and Naik note that the Global Advisory Committee on Vaccine Safety has documented many deaths in children with pre-existing heart disease after they were administered the pentavalent vaccine (combined diphtheria, tetanus, pertussis, Hib, and hepatitis-B vaccine).
READ MORE
From stuff.co.nz
2009
The family of a Mataura possum hunter who died from 1080 poisoning 43 years ago want his death publicly acknowledged by the Department of Conservation and Animal Health Board.
Both agencies say no-one has died of accidental 1080 poisoning in New Zealand.
But Ian Buchanan’s widow, 69-year-old Elva Wicks, of Invercargill, said that was not true.
Wicks was 25 years old and the mother of three young children when her first husband, Ian Buchanan, died a “ghastly death” in Dunedin Hospital in 1966.
Coroner J Murray found Buchanan died from “central nervous system depression accompanied by respiratory and cardiac failure due to 1080 poisoning”.
The poison was found by investigators in a tin of jam kept in a back porch cupboard, but it was never discovered how the poison came to be in the jam, or how Buchanan consumed the 1080.
“If DOC and the AHB say they haven’t heard anything about Ian’s death it’s because they don’t want to hear,” Wicks said.
“It is documented on his death certificate – how can they deny it?”
AHB spokeswoman Anne McLean said the manner of Buchanan’s death was unclear.
DOC spokesman Rory Newsam said the department was unaware of Buchanan’s death until alerted by the media this week.
SOURCE
http://www.stuff.co.nz/the-press/news/3097688/Hunters-family-in-1080-battle
